Lifeboat Foundation

Stress sensitive neural circuits change the gut microbiome and the duodenal glands.

Precision mapping of a gut-brain circuit w/ functional impact.

Glands in the small intestine (Brunner’s) have GLP-1 receptors, are stress-sensitive, connect to the brain via the vagal nerve, modulate bacterial…

Continue reading “How the stressed-out brain can weaken the immune system” »

The neural representations of a perceived image and the memory of it are almost the same. New work shows how and why they are different.

Microbe found in cat poo could be harnessed to deliver large, complex proteins across the blood–brain barrier.

As a medical scientist, he looks at how new findings will help develop new treatments to revive people now considered hopeless and restore brain damage.

Strengthening Public Health Systems For Healthier And Longer Lives — Dr. Ashwin Vasan, Commissioner, NYC Department of Health and Mental Hygiene.

Dr. Ashwin Vasan, MD, PhD is the Commissioner of the New York City Department of Health and Mental Hygiene (https://www.nyc.gov/site/doh/about/ab…).

Continue reading “Dr. Ashwin Vasan — Commissioner — NYC Dept. of Health & Mental Hygiene — Strengthening Public Health” »

Scientists propose that human consciousness evolved primarily for social survival, enabling better communication and interaction within communities, rather than just individual benefit, challenging traditional views on the evolution of subjective awareness.

Brain-computer interfaces (BCIs) can translate a person’s brainwaves into action, but these devices are vulnerable to hacking.

Researchers led by the University of California, Irvine have discovered how the TREM2 R47H genetic mutation causes certain brain areas to develop abnormal protein clumps, called beta-amyloid plaques, associated with late-onset Alzheimer’s disease. Leveraging single-cell Merfish spatial transcriptomics technology, the team was able to profile the effects of the mutation across multiple cortical and subcortical brain regions, offering first-of-their-kind insights at the single-cell level.

The study, published in Molecular Psychiatry, compared the brains of normal mice and special mouse models that undergo changes like those in humans with Alzheimer’s.

Findings revealed that the TREM2 mutation led to divergent patterns of beta-amyloid plaque accumulation in various parts of the brain involved in higher-level functions such as memory, reasoning and speech. It also affected certain cell types and their gene expression near the plaques.

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